A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency

Immunology and Microbiology

• Human Immunodeficiency Virus 100%
• Mutation 83%
• Tumor Necrosis Factor Alpha 50%
• Long Terminal Repeat 50%
• Cells 50%
• Point Mutation 33%
• Wild Type 16%
• Phenotype 16%
• Cloning 16%
• Transactivation 16%
• Transient Transfection 16%
• Experiment 16%
• Clone 16%
• Base Pairing 16%
• Virus 16%

Neuroscience

• HIV 100%
• Tumor Necrosis Factor Alpha 50%
• Long Terminal Repeat 50%
• Complementary DNA 33%
• Point Mutation 33%
• Phenotype 16%
• Acetate 16%
• Phorbol 16%
• Transactivation 16%
• Base Pairing 16%
• Transient Transfection 16%
• Cloning 16%
• Protein 16%